TY  - JOUR
A1  - Cabrera-Fuentes, Hector A.
A1  - Aragones, Julian
A1  - Bernhagen, Jürgen
A1  - Boening, Andreas
A1  - Boisvert, William A.
A1  - Bøtker, Hans E.
A1  - Bulluck, Heerajnarain
A1  - Cook, Stuart
A1  - Di Lisa, Fabio
A1  - Engel, Felix B.
A1  - Engelmann, Bernd
A1  - Ferrazzi, Fulvia
A1  - Ferdinandy, Peter
A1  - Fong, Alan
A1  - Fleming, Ingrid
A1  - Gnaiger, Erich
A1  - Hernández-Reséndiz, Sauri
A1  - Kalkhoran, Siavash Beikoghli
A1  - Kim, Moo Hyun
A1  - Lecour, Sandrine
A1  - Liehn, Elisa A.
A1  - Marber, Michael S.
A1  - Mayr, Manuel
A1  - Miura, Tetsuji
A1  - Ong, Sang-Bing
A1  - Peter, Karlheinz
A1  - Sedding, Daniel
A1  - Singh, Manvendra K.
A1  - Suleiman, M. Saadeh
A1  - Schnittler, Hans J.
A1  - Schulz, Rainer
A1  - Shim, Winston
A1  - Tello, Daniel
A1  - Vogel, Carl-Wilhelm
A1  - Walker, Malcolm
A1  - Li, Qilong Oscar Yang
A1  - Yellon, Derek M.
A1  - Hausenloy, Derek J.
A1  - Preissner, Klaus T.
T1  - From basic mechanisms to clinical applications in heart protection, new players in cardiovascular diseases and cardiac theranostics: meeting report from the third international symposium on "New frontiers in cardiovascular research"
T2  - Basic research in cardiology
N2  - In this meeting report, particularly addressing the topic of protection of the cardiovascular system from ischemia/reperfusion injury, highlights are presented that relate to conditioning strategies of the heart with respect to molecular mechanisms and outcome in patients’ cohorts, the influence of co-morbidities and medications, as well as the contribution of innate immune reactions in cardioprotection. Moreover, developmental or systems biology approaches bear great potential in systematically uncovering unexpected components involved in ischemia–reperfusion injury or heart regeneration. Based on the characterization of particular platelet integrins, mitochondrial redox-linked proteins, or lipid-diol compounds in cardiovascular diseases, their targeting by newly developed theranostics and technologies opens new avenues for diagnosis and therapy of myocardial infarction to improve the patients’ outcome.
KW  - Cardiomyocyte signaling pathways
KW  - Cardioprotection
KW  - Cardiovascular disease
KW  - Comorbidities
KW  - Drug targeting
KW  - Endothelial permeability
KW  - Extracellular RNA (eRNA)
KW  - Heart regeneration
KW  - Induced pluripotent stem cells
KW  - Ischemia–reperfusion injury
KW  - Lipid metabolism
KW  - MicroRNAs (miRNAs)
KW  - Mitochondria
KW  - Remote ischemic conditioning
Y1  - 2016
UR  - http://publikationen.ub.uni-frankfurt.de/frontdoor/index/index/docId/42737
UR  - https://nbn-resolving.org/urn:nbn:de:hebis:30:3-427376
SN  - 1435-1803
N1  - This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.
VL  - 111
IS  - 69
SP  - 1
EP  - 13
PB  - Springer
CY  - Heidelberg
ER  -