TY - JOUR A1 - Petsos, Hari A1 - Schacher, Beate A1 - Ramich, Tatjana A1 - Nickles, Katrin A1 - Dannewitz, Bettina A1 - Arendt, Susanne A1 - Seidel, Kathrin A1 - Eickholz, Peter T1 - Retrospectively analysed tooth loss in periodontally compromised patients: long-term results 10 years after active periodontal therapy - patient-related outcomes T2 - Journal of periodontal research N2 - Background and Objective: Long-term tooth retention is the ultimate goal of periodontal therapy. Aim of this study was to evaluate tooth loss (TL) during 10 years of supportive periodontal therapy (SPT) in periodontal compromised patients and to identify factors influencing TL on patient level. Material and Methods: Patients were re-examined 120 ± 12 months after active periodontal therapy. TL and risk factors [smoking, initial diagnosis, SPT adherence, interleukin-1 polymorphism, cardiovascular diseases, age at baseline, bleeding on probing (BOP), change of practitioner, insurance status, number of SPT, marital and educational status] influencing TL on patient level were assessed. Results: One-hundred patients (52 female, mean age 65.6 ± 11 years) lost 121 of 2428 teeth (1.21 teeth/patient; 0.12 teeth/patient/y) during 10 years of SPT. Forty-two of these were lost for periodontal reasons (0.42 teeth/patient; 0.04 teeth/patient/y). Significantly more teeth were lost due to other reasons (P < .001). Smoking, baseline severity of periodontitis, non-adherent SPT, positive interleukin-1 polymorphism, marital and educational status, private insurance, older age at baseline and BOP, small number of SPT were identified as patient-related risk factors for TL (P < .05). Conclusion: During 120 ± 12 months of SPT, only a small number of teeth was lost in periodontally compromised patients showing the positive effect of a well-established periodontal treatment concept. The remaining risk for TL should be considered using risk-adopted SPT allocation. KW - long-term success KW - periodontal risk factors KW - supportive periodontal therapy KW - tooth loss Y1 - 2020 UR - http://publikationen.ub.uni-frankfurt.de/frontdoor/index/index/docId/63843 UR - https://nbn-resolving.org/urn:nbn:de:hebis:30:3-638430 SN - 1600-0765 N1 - The study was self-funded by the authors and their institutions. Hain Lifescience GmbH (Nehren, Germany) supported analyses for IL-1 polymorphism. VL - 55 IS - 6 SP - 946 EP - 958 PB - Wiley-Blackwell CY - Oxford [u.a.] ER -