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We study the beam-energy and system-size dependence of \phi meson production (using the hadronic decay mode \phi -- K+K-) by comparing the new results from Cu+Cu collisions and previously reported Au+Au collisions at \sqrt{s_NN} = 62.4 and 200 GeV measured in the STAR experiment at RHIC. Data presented are from mid-rapidity (|y|<0.5) for 0.4 < pT < 5 GeV/c. At a given beam energy, the transverse momentum distributions for \phi mesons are observed to be similar in yield and shape for Cu+Cu and Au+Au colliding systems with similar average numbers of participating nucleons. The \phi meson yields in nucleus-nucleus collisions, normalised by the average number of participating nucleons, are found to be enhanced relative to those from p+p collisions with a different trend compared to strange baryons. The enhancement for \phi mesons is observed to be higher at \sqrt{s_NN} = 200 GeV compared to 62.4 GeV. These observations for the produced \phi(s\bar{s}) mesons clearly suggest that, at these collision energies, the source of enhancement of strange hadrons is related to the formation of a dense partonic medium in high energy nucleus-nucleus collisions and cannot be alone due to canonical suppression of their production in smaller systems.
The STAR Collaboration at the Relativistic Heavy Ion Collider presents measurements of 𝐽/𝜓→𝑒+𝑒− at midrapidity and high transverse momentum (𝑝𝑇>5 GeV/𝑐) in 𝑝+𝑝 and central Cu+Cu collisions at √𝑠𝑁𝑁=200 GeV. The inclusive 𝐽/𝜓 production cross section for Cu+Cu collisions is found to be consistent at high 𝑝𝑇 with the binary collision-scaled cross section for 𝑝+𝑝 collisions. At a confidence level of 97%, this is in contrast to a suppression of 𝐽/𝜓 production observed at lower 𝑝𝑇. Azimuthal correlations of 𝐽/𝜓 with charged hadrons in 𝑝+𝑝 collisions provide an estimate of the contribution of 𝐵-hadron decays to 𝐽/𝜓 production of 13%±5%.
Ten South American species are removed from the genus Odontocera Audinet-Serville (Coleoptera: Cerambycidae) and placed in Odontocroton Clarke new genus. The new genus is further organized into two informal groups. Group A includes Odontocroton flavicauda (Bates, 1873) new combination, Odontocroton flavirostris (Melzer, 1930) new combination, Odontocroton melzeri (Fisher, 1952) new combination and Odontocroton soror (Gounelle, 1911) new combination. Group B includes Odontocroton apicalis (Klug, 1825) new combination, Odontocroton quinquecallosus (Zajciw, 1963) new combination, Odontocroton sanguinolentus (Bates, 1873) new combination, Odontocroton septemtuberculatus (Zajciw, 1963) new combination, Odontocroton rufifrons (Fisher, 1937) new rank and new combination, and provisionally Odontocroton monnei (Zajciw, 1968), new combination. A monotypic new genus, Rhinobatesia Clarke, is described for the Central American species Rhinobatesia rugicollis (Bates, 1880) new combination, which was formerly in Odontocera. The Central American Odontocera nevermanni Fisher, 1930 is placed as a junior synonym of R. rugicollis, and Odontocera typhoeus Fisher, 1947 is placed as a junior synonym of Odontogracilis gracilis (Klug, 1825). A key to separate Odontocroton and Rhinobatesia as well as the species of the former is provided. All species are illustrated, including the tegmen of the aedeagus when available. Host flower records for the Bolivian species are also provided.
Background: Alzheimer's disease is a common debilitating dementia with known heritability, for which 20 late onset susceptibility loci have been identified, but more remain to be discovered. This study sought to identify new susceptibility genes, using an alternative gene-wide analytical approach which tests for patterns of association within genes, in the powerful genome-wide association dataset of the International Genomics of Alzheimer's Project Consortium, comprising over 7 m genotypes from 25,580 Alzheimer's cases and 48,466 controls.
Principal findings: In addition to earlier reported genes, we detected genome-wide significant loci on chromosomes 8 (TP53INP1, p = 1.4×10−6) and 14 (IGHV1-67 p = 7.9×10−8) which indexed novel susceptibility loci.
Significance: The additional genes identified in this study, have an array of functions previously implicated in Alzheimer's disease, including aspects of energy metabolism, protein degradation and the immune system and add further weight to these pathways as potential therapeutic targets in Alzheimer's disease.