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In works of Maghrebi authors like Tahar Ben Jelloun (Morocco), the body is the central medium that generates and constitutes the narration. The authors stand in the tradition of oral folk literature, which increasingly has been displaced by French written literature. Hence there is a tendency in postcolonial Maghrebi texts to reintegrate the performative act of narrating via the body into the literary structures of the novels. This becomes manifest in poly-phonic and poly-perspectival narrative experiments in which, with recourse to the halqa (the typical oral narrative situation), a re-territorialization (Deleuze/Guattari) of the body is performed.
In this context the body in literature plays a central role on the level of the metadiegesis: it is presented as the medium of narration. Using as an example Tahar Ben Jellouns novel L’enfant de sable (1985), the aim of this essay is to show how halqa elements and narrative influences from The Arabian Nights structure the text, which becomes a hybrid between medium and embodiment (Fischer-Lichte) by simulating eventfulness.
On the level of the diegesis, the body plays likewise a decisive role as subject of the storyline, becoming the most important medium for the expression of emotions, thoughts or attitudes. Body language is deliberately utilized by the authors to discuss ways of dealing with traditions, the negotiation of social relations and the (de-)construction of identity. Social order, power structures, hierarchies, existing values and norms are communicated and constituted via body language.
Recent studies suggest that synaptic lysophosphatidic acids (LPAs) augment glutamate-dependent cortical excitability and sensory information processing in mice and humans via presynaptic LPAR2 activation. Here, we studied the consequences of LPAR2 deletion or antagonism on various aspects of cognition using a set of behavioral and electrophysiological analyses. Hippocampal neuronal network activity was decreased in middle-aged LPAR2−/− mice, whereas hippocampal long-term potentiation (LTP) was increased suggesting cognitive advantages of LPAR2−/− mice. In line with the lower excitability, RNAseq studies revealed reduced transcription of neuronal activity markers in the dentate gyrus of the hippocampus in naïve LPAR2−/− mice, including ARC, FOS, FOSB, NR4A, NPAS4 and EGR2. LPAR2−/− mice behaved similarly to wild-type controls in maze tests of spatial or social learning and memory but showed faster and accurate responses in a 5-choice serial reaction touchscreen task requiring high attention and fast spatial discrimination. In IntelliCage learning experiments, LPAR2−/− were less active during daytime but normally active at night, and showed higher accuracy and attention to LED cues during active times. Overall, they maintained equal or superior licking success with fewer trials. Pharmacological block of the LPAR2 receptor recapitulated the LPAR2−/− phenotype, which was characterized by economic corner usage, stronger daytime resting behavior and higher proportions of correct trials. We conclude that LPAR2 stabilizes neuronal network excitability upon aging and allows for more efficient use of resting periods, better memory consolidation and better performance in tasks requiring high selective attention. Therapeutic LPAR2 antagonism may alleviate aging-associated cognitive dysfunctions.