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The present paper aims at providing empirical evidence for dialectal variation concerning the perception of the central vowel [ɐ] in European Portuguese (EP). More concretely, this study compares the perception of the contrast between [a] and [ɐ] by native speakers of two varieties of EP: 23 speakers of a northern Portuguese dialect (from the city of Braga) and 23 speakers of the Littoral Center variety of EP (from the city of Lisbon, defined as Standard European Portuguese (SEP)). Based on a discrimination test, the results show that the two groups of speakers differ with respect to the perception of the contrast between the two central vowels under investigation. The speakers of the northern variety differentiate less between the two central vowels compared to the speakers from Lisbon.
Diese Arbeit untersucht die Perzeption der Vokale /a/, /ɐ/ und /ɐ̃/ durch deutsche erwachsene Portugiesischlerner. Ziel war herauszufinden, ob deutsche L1-Sprecher in der Lage sind, die genannten Laute zu unterscheiden, unter anderem in Abhängigkeit von ihrem Fremdsprachenniveau im EP.
Die Teilnehmer der Studie waren 27 deutsche Muttersprachler, die EP im Erwachsenenalter und zumeist an der Universität erlernen oder erlernt haben. Die Kontrollgruppe bildeten 33 portugiesische Muttersprachler. Beide Gruppen nahmen an einem Online-Perzeptionstest teil, dessen Aufbau sich an der Studie von Darcy und Krüger (2012) orientiert. Während des Tests mussten die Probanden aus einer Lautsequenz von drei einsilbigen Kunstwörtern mit der Silbenstruktur CVC dasjenige Kunstwort identifizieren, das einen anderen Vokal beinhaltete als die anderen zwei. Neben dem Perzeptionstest nahmen alle Teilnehmer an einem Einstufungstest für das EP teil, der aus einem C-Test und einem Vokabeltest bestand.
The genetic make-up of an individual contributes to the susceptibility and response to viral infection. Although environmental, clinical and social factors have a role in the chance of exposure to SARS-CoV-2 and the severity of COVID-191,2, host genetics may also be important. Identifying host-specific genetic factors may reveal biological mechanisms of therapeutic relevance and clarify causal relationships of modifiable environmental risk factors for SARS-CoV-2 infection and outcomes. We formed a global network of researchers to investigate the role of human genetics in SARS-CoV-2 infection and COVID-19 severity. Here we describe the results of three genome-wide association meta-analyses that consist of up to 49,562 patients with COVID-19 from 46 studies across 19 countries. We report 13 genome-wide significant loci that are associated with SARS-CoV-2 infection or severe manifestations of COVID-19. Several of these loci correspond to previously documented associations to lung or autoimmune and inflammatory diseases3,4,5,6,7. They also represent potentially actionable mechanisms in response to infection. Mendelian randomization analyses support a causal role for smoking and body-mass index for severe COVID-19 although not for type II diabetes. The identification of novel host genetic factors associated with COVID-19 was made possible by the community of human genetics researchers coming together to prioritize the sharing of data, results, resources and analytical frameworks. This working model of international collaboration underscores what is possible for future genetic discoveries in emerging pandemics, or indeed for any complex human disease.