Refine
Year of publication
Document Type
- Preprint (103)
- Article (70)
- Conference Proceeding (15)
- Contribution to a Periodical (1)
- Review (1)
Has Fulltext
- yes (190)
Is part of the Bibliography
- no (190) (remove)
Keywords
- Kollisionen schwerer Ionen (16)
- heavy ion collisions (14)
- UrQMD (6)
- QGP (5)
- Quark-Gluon-Plasma (5)
- Quark Gluon Plasma (4)
- Drell-Yan (3)
- Hadron (3)
- Heavy-ion collisions (3)
- MEMOs (3)
Institute
The QGP that might be created in ultrarelativistic heavy-ion collisions is expected to radiate thermal dilepton radiation. However, this thermal dilepton radiation interferes with dileptons originating from hadron decays. In the invariant mass region between the f and J=y peak (1GeV <= M l+l <=. 3GeV) the most substantial background of hadron decays originates from correlated DD¯ -meson decays. We evaluate this background using a Langevin simulation for charm quarks. As background medium we utilize the well-tested UrQMD-hybrid model. The required drag and diffusion coefficients are taken from a resonance approach. The decoupling of the charm quarks from the hot medium is performed at a temperature of 130MeV and as hadronization mechanism a coalescence approach is chosen. This model for charm quark interactions with the medium has already been successfully applied to the study of the medium modification and the elliptic flow at FAIR, RHIC and LHC energies. In this proceeding we present our results for the dilepton radiation from correlated D¯D decays at RHIC energy in comparison to PHENIX measurements in the invariant mass range between 1 and 3 GeV using different interaction scenarios. These results can be utilized to estimate the thermal QGP radiation.
As microscopic transport models usually have difficulties to deal with in-medium effects in heavy-ion collisions, we present an alternative approach that uses coarse-grained output from transport calculations with the UrQMD model to determine thermal dilepton emission rates. A four-dimensional space-time grid is set up to extract local baryon and energy densities, respectively temperature and baryon chemical potential. The lepton pair emission is then calculated for each cell of the grid using thermal equilibrium rates. In the current investigation we inlcude the medium-modified r spectral function by Eletsky et al., as well as contributions from the QGP and four-pion interactions for high collision energies. First dielectron invariant mass spectra for Au+Au collisions at 1.25 AGeV and for dimuons from In+In at 158 AGeV are shown. At 1.25 AGeV a clear enhancement of the total dilepton yield as compared to a pure transport result is observed. In the latter case, we compare our outcome with the NA60 dimuon excess data. Here a good agreement is achieved, but the yield in the low-mass tail is underestimated. In general the results show that the coarse-graining approach gives reasonable results and can cover a broad collision-energy range.
We analyze hadrochemical freeze-out in central Pb+Pb collisions at CERN SPS and LHC energies. Employing the UrQMD hybrid transport model we study the effects of the final hadron/resonance expansion phase on the hadron multiplicities established at hadronization. The bulk meson yields freeze out directly at hadronization whereas the baryon-antibaryon sector is subject to significant alterations, due to annihilation and regeneration processes. We quantify the latter changes by survival factors for each species which are applied to modify the statistical model predictions for the data. The modified SM analysis recovers the hadronization points, which coincide with the recent lattice QCD predictions of the parton-hadron transition line at finite baryochemical potential.
Recent results on baryon production in relativistic heavy ion collisions show that a revision of the chemical freeze-out conditions is necessary. Particularly, there is evidence that chemical freezeout does not occur at full chemical equilibrium. We present a method to reconstruct original hadronization conditions and show that the newly found points in the T − µB plane are in very good agreement with extrapolations of the lattice QCD critical line.
Tumour cells show a varying susceptibility to radiation damage as a function of the current cell cycle phase. While this sensitivity is averaged out in an unperturbed tumour due to unsynchronised cell cycle progression, external stimuli such as radiation or drug doses can induce a resynchronisation of the cell cycle and consequently induce a collective development of radiosensitivity in tumours. Although this effect has been regularly described in experiments it is currently not exploited in clinical practice and thus a large potential for optimisation is missed. We present an agent-based model for three-dimensional tumour spheroid growth which has been combined with an irradiation damage and kinetics model. We predict the dynamic response of the overall tumour radiosensitivity to delivered radiation doses and describe corresponding time windows of increased or decreased radiation sensitivity. The degree of cell cycle resynchronisation in response to radiation delivery was identified as a main determinant of the transient periods of low and high radiosensitivity enhancement. A range of selected clinical fractionation schemes is examined and new triggered schedules are tested which aim to maximise the effect of the radiation-induced sensitivity enhancement. We find that the cell cycle resynchronisation can yield a strong increase in therapy effectiveness, if employed correctly. While the individual timing of sensitive periods will depend on the exact cell and radiation types, enhancement is a universal effect which is present in every tumour and accordingly should be the target of experimental investigation. Experimental observables which can be assessed non-invasively and with high spatio-temporal resolution have to be connected to the radiosensitivity enhancement in order to allow for a possible tumour-specific design of highly efficient treatment schedules based on induced cell cycle synchronisation.
Author Summary: The sensitivity of a cell to a dose of radiation is largely affected by its current position within the cell cycle. While under normal circumstances progression through the cell cycle will be asynchronous in a tumour mass, external influences such as chemo- or radiotherapy can induce a synchronisation. Such a common progression of the inner clock of the cancer cells results in the critical dependence on the effectiveness of any drug or radiation dose on a suitable timing for its administration. We analyse the exact evolution of the radiosensitivity of a sample tumour spheroid in a computer model, which enables us to predict time windows of decreased or increased radiosensitivity. Fractionated radiotherapy schedules can be tailored in order to avoid periods of high resistance and exploit the induced radiosensitivity for an increase in therapy efficiency. We show that the cell cycle effects can drastically alter the outcome of fractionated irradiation schedules in a spheroid cell system. By using the correct observables and continuous monitoring, the cell cycle sensitivity effects have the potential to be integrated into treatment planing of the future and thus to be employed for a better outcome in clinical cancer therapies.
Direct photon emission in heavy-ion collisions is calculated within a relativistic micro+macro
hybrid model and compared to the microscopic transport model UrQMD. In the hybrid approach,
the high-density part of the collision is calculated by an ideal 3+1-dimensional hydrodynamic
calculation, while the early (pre-equilibrium-) and late (rescattering-) phase are calculated with
the transport model. Different scenarios of the transition from the macroscopic description to
the transport model description and their effects are studied. The calculations are compared to
measurements by the WA98-collaboration and predictions for the future CBM-experiment are
made.
We explore the shape and orientation of the freezeout region of non-central heavy ion collisions.
For this we fit the freezeout distribution with a tilted ellipsoid. The resulting tilt angle is compared
to the same tilt angle extracted via an azimuthally sensitive HBT analysis. This allows to access
the tilt angle experimentally, which is not possible directly from the freezeout distribution. We
also show a systematic study on the system decoupling time dependence on dNch/dh, using HBT
results from the UrQMD transport model. In this study we found that the decoupling time scales
with (dNch/dh)1/3 within each energy, but the scaling is broken across energies.
The biological effects of energetic heavy ions are attracting increasing interest for their applications in cancer therapy and protection against space radiation. The cascade of events leading to cell death or late effects starts from stochastic energy deposition on the nanometer scale and the corresponding lesions in biological molecules, primarily DNA. We have developed experimental techniques to visualize DNA nanolesions induced by heavy ions. Nanolesions appear in cells as “streaks” which can be visualized by using different DNA repair markers. We have studied the kinetics of repair of these “streaks” also with respect to the chromatin conformation. Initial steps in the modeling of the energy deposition patterns at the micrometer and nanometer scale were made with MCHIT and TRAX models, respectively.
We present results on Hanbury Brown-Twiss (HBT) radii extracted from the Ultra-relativistic Molecular Dynamics (UrQMD) approach to relativistic heavy ion collisions. The present investigation provides a comparison of results from pure hadronic transport calculations to a Boltzmann + Hydrodynamic hybrid approach with an intermediate hydrodynamic phase. For the hydrodynamic phase different Equations of State (EoS) have been employed, i.e. bag model, hadron resonance gas and a chiral EoS. The influence of various freeze-out scenarios has been investigated and shown to be negligible if hadronic rescatterings after the hydrodynamic evolution are included. Furthermore, first results of the source tilt from azimuthal sensitive HBT and the direct extraction from the transport model are presented and exhibit a very good agreement with E895 data at AGS.
A mechanism for locally density-dependent dynamic parton rearrangement and fusion has been implemented into the Ultrarelativistic Quantum Molecular Dynamics (UrQMD) approach. The same mechanism has been previously built in the Quark Gluon String Model (QGSM). This rearrangement and fusion approach based on parton coalescence ideas enables the description of multi-particle interactions, namely 3 -> 3 and 3 -> 2, between (pre)hadronic states in addition to standard binary interactions. The UrQMD model (v2.3) extended by these additional processes allows to investigate implications of multi-particle interactions on the reaction dynamics of ultrarelativistic heavy ion collisions. The mechanism, its implementation and first results of this investigation are presented and discussed.