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Revision of Hawaiian, Australasian, Oriental, and Japanese Parandrinae (Coleoptera, Cerambycidae)
(2010)
A comprehensive revision of the Subfamily Parandrinae (Coleoptera, Cerambycidae) from the Hawaiian, Australasian, Oriental, and Japanese regions is presented. Seven (7) new genera are described: Komiyandra, Melanesiandra, Papuandra, Storeyandra, Hawaiiandra, Caledonandra, and Malukandra. All known, indigenous species from these regions are assigned to new genera resulting in the following new combinations: Komiyandra janus (Bates, 1875), K. shibatai (Hayashi, 1963), K. formosana (Miwa and Mitono, 1939), K. lanyuana (Hayashi, 1981), Melanesiandra striatifrons (Fairmaire, 1879), M. solomonensis (Arigony, 1983), Caledonandra austrocaledonica (Montrouzier, 1861), C. passandroides (Thomson, 1867), Hawaiiandra puncticeps (Sharp, 1878), Malukandra heterostyla (Lameere, 1902), Storeyandra frenchi (Blackburn, 1895), and Papuandra araucariae (Gressitt, 1959). Thirty-one (31) new species are described: Komiyandra javana, K. nayani, K. ohbayashii, K. luzonica, K. philippinensis, K. mindanao, K. mehli, K. vivesi, K. lombokia, K. sulawesiana, K. irianjayana, K. menieri, K. sangihe, K. mindoro, K. niisatoi, K. drumonti, K. cabigasi, K. koni, K. johkii, K. poggii, K. uenoi, Melanesiandra bougainvillensis, M. birai, Papuandra gressitti, P. weigeli, P. queenslandensis, P. norfolkensis, P. rothschildi, P. oberthueri, Malukandra jayawijayana and M. hornabrooki. A lectotype is designated for Parandra janus Bates, 1875. Komiyandra janus (Bates, 1875) is excluded from nearly all previously reported locations, even one location given in the original description, and is now only known from Sulawesi. A paralectotype of Parandra janus Bates, 1875, is designated as a paratype for Komiyandra menieri, new species. Komiyandra formosana is excluded from the Japanese (Ryukyu Is.) fauna. Parandra vitiensis Nonfried, 1894, is again placed in synonymy with P. striatifrons Fairmaire (now Melanesiandra striatifrons). A neotype is designated for Parandra austrocaledonica Montrouzier, 1861. A lectotype is designated for Parandra janus Bates, 1875. The lectotype of Parandra gabonica Thomson, 1858, designated by Quentin and Villiers (1975) is considered invalid. Papuandra araucariae (Gressitt, 1959) is excluded from the fauna of Norfolk Island. The African species Stenandra kolbei (Lameere, 1903) is reported for the first time from Asia (N. Vietnam). Keys are presented to separate worldwide genera of Parandrini and all species within the study regions. Illustrations are provided for all species including many special characters to differentiate genera and species.
When a nanoparticle is irradiated by an intense laser pulse, it turns into a nanoplasma, a transition that is accompanied by many interesting nonequilibrium dynamics. So far, most experiments on nanoplasmas use ion measurements, reflecting the outside dynamics in the nanoparticle. Recently, the direct observation of the ultrafast structural dynamics on the inside of the nanoparticle also became possible with the advent of x-ray free electron lasers (XFELs). Here, we report on combined measurements of structural dynamics and speeds of ions ejected from nanoplasmas produced by intense near-infrared laser irradiations, with the control of the initial plasma conditions accomplished by widely varying the laser intensity (9×1014 W/cm2 to 3×1016 W/cm2). The structural change of nanoplasmas is examined by time-resolved x-ray diffraction using an XFEL, while the kinetic energies of ejected ions are measured by an ion time-of-fight method under the same experimental conditions. We find that the timescale of crystalline disordering in nanoplasmas strongly depends on the laser intensity and scales with the inverse of the average speed of ions ejected from the nanoplasma. The observations support a recently suggested scenario for nanoplasma dynamics in the wide intensity range, in which crystalline disorder in nanoplasmas is caused by a rarefaction wave propagating at a speed comparable with the average ion speed from the surface toward the inner crystalline core. We demonstrate that the scenario is also applicable to nanoplasma dynamics in the hard x-ray regime. Our results connect the outside nanoplasma dynamics to the loss of structure inside the sample on the femtosecond timescale.
The genetic make-up of an individual contributes to the susceptibility and response to viral infection. Although environmental, clinical and social factors have a role in the chance of exposure to SARS-CoV-2 and the severity of COVID-191,2, host genetics may also be important. Identifying host-specific genetic factors may reveal biological mechanisms of therapeutic relevance and clarify causal relationships of modifiable environmental risk factors for SARS-CoV-2 infection and outcomes. We formed a global network of researchers to investigate the role of human genetics in SARS-CoV-2 infection and COVID-19 severity. Here we describe the results of three genome-wide association meta-analyses that consist of up to 49,562 patients with COVID-19 from 46 studies across 19 countries. We report 13 genome-wide significant loci that are associated with SARS-CoV-2 infection or severe manifestations of COVID-19. Several of these loci correspond to previously documented associations to lung or autoimmune and inflammatory diseases3,4,5,6,7. They also represent potentially actionable mechanisms in response to infection. Mendelian randomization analyses support a causal role for smoking and body-mass index for severe COVID-19 although not for type II diabetes. The identification of novel host genetic factors associated with COVID-19 was made possible by the community of human genetics researchers coming together to prioritize the sharing of data, results, resources and analytical frameworks. This working model of international collaboration underscores what is possible for future genetic discoveries in emerging pandemics, or indeed for any complex human disease.